New HSP gene discovered – SPTSSA

Found to cause complex HSP in 3 children

Studies in patient skin cells, human kidney cells and in fruit flies show that variants in the SPTSSA gene cause imbalance in lipid production and regulation, and are responsible for impaired early brain development and function.

Abstract

Sphingolipids are a diverse family of lipids with critical structural and signalling functions in the mammalian nervous system, where they are abundant in myelin membranes. Serine palmitoyltransferase, the enzyme that catalyses the rate-limiting reaction of sphingolipid synthesis, is comprised of multiple subunits including an activating subunit, SPTSSA. Sphingolipids are both essential and cytotoxic and their synthesis must therefore be tightly regulated. Key to the homeostatic regulation is the ORMDL proteins that are bound to serine palmitoyltransferase and mediate feedback inhibition of enzymatic activity when sphingolipid levels become excessive.

Exome sequencing identified potential disease-causing variants in SPTSSA in three children presenting with a complex form of hereditary spastic paraplegia. The effect of these variants on the catalytic activity and homeostatic regulation of serine palmitoyltransferase was investigated in human embryonic kidney cells, patient fibroblasts and Drosophila. Our results showed that two different pathogenic variants in SPTSSA caused a hereditary spastic paraplegia resulting in progressive motor disturbance with variable sensorineural hearing loss and language/cognitive dysfunction in three individuals. The variants in SPTSSA impaired the negative regulation of serine palmitoyltransferase by ORMDLs leading to excessive sphingolipid synthesis based on biochemical studies and in vivo studies in Drosophila.

These findings support the pathogenicity of the SPTSSA variants and point to excessive sphingolipid synthesis due to impaired homeostatic regulation of serine palmitoyltransferase as responsible for defects in early brain development and function.

SOURCE:  Brain. 2023 Jan 30;awac460. doi: 10.1093/brain/awac460. Online ahead of print. PMID: 36718090 Published by Oxford University Press on behalf of the Guarantors of Brain 2023.

SPTSSA variants alter sphingolipid synthesis and cause a complex hereditary spastic paraplegia

Siddharth Srivastava  1 Hagar Mor Shaked  2 Kenneth Gable  3 Sita D Gupta  3 Xueyang Pan  4   5 Niranjanakumari Somashekarappa  3 Gongshe Han  3 Payam Mohassel  6 Marc Gotkine  2 Elizabeth Doney  7 Paula Goldenberg  8 Queenie K G Tan  9 Yi Gong  10   11 Benjamin Kleinstiver  11   12   13 Brian Wishart  14 Heidi Cope  9 Claudia Brito Pires  10   11 Hannah Stutzman  11   12 Rebecca C Spillmann  9 Undiagnosed Disease NetworkReza Sadjadi  10 Orly Elpeleg  2 Chia-Hsueh Lee  15 Hugo J Bellen  4   5 Simon Edvardson  16 Florian Eichler  10   11 Teresa M Dunn  3   

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1. Department of Neurology, Rosamund Stone Zander Translational Neuroscience Center, Boston Children’s Hospital, Harvard Medical School, Boston, MA 02115, USA.

2. Department of Genetics, Hadassah Medical Center and Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem 91120, Israel.

3. Department of Biochemistry and Molecular Biology, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA.

4. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.

5. Jan and Dan Duncan Neurological Research Institute, Texas Children’s Hospital, Houston, TX 77030, USA.

6. Neuromuscular and Neurogenetic Disorders of Childhood Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20814, USA.

7. Massachusetts Eye and Ear, Boston, MA 02114, USA.

8. Department of Pediatrics, Section on Medical Genetics, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.

9. Department of Pediatrics, Division of Medical Genetics, Duke University School of Medicine, Durham, NC 27710, USA.

10. Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.

11. Center for Genomic Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.

12. Department of Pathology, Massachusetts General Hospital, Boston, MA 02114, USA.

13. Department of Pathology, Harvard Medical School, Boston, MA 02115, USA.

14. Physical Medicine and Rehabilitation, Spaulding Rehabilitation Hospital, Harvard Medical School, Boston, MA 02114, USA.

15. Department of Structural Biology, St. Jude Children’s Research Hospital, Memphis, TN 38105, USA.

16. Pediatric Neurology Unit, Hadassah University Hospital, Mount Scopus, Jerusalem 91240, Israel.

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